Current neuroscience does not support a simple “chemical imbalance” model with “normal” ranges the way blood labs work. Dopamine and norepinephrine signaling are involved, but ADHD is better understood as differences in how certain brain networks develop and regulate behavior, particularly fronto-striatal and limbic circuits. It’s about signal timing, regulation, and responsiveness- not just having too little or too much of a neurotransmitter.
Likewise, your claims about a “smaller frontal lobe” are oversimplified. Imaging studies show average developmental and connectivity differences in regions like the prefrontal cortex, basal ganglia, and limbic system, but these differences are subtle, highly variable, and strongly overlapping with neurotypical brains. They are not defects and not diagnostic on their own.
Framing this as an adaptation rather than a defect is more than reasonable. Traits associated with ADHD, namely novelty seeking, rapid scanning of the environment, high energy, risk tolerance, and creative problem-solving offered advantages in ancestral high-stimulus environments. What you define as a disorder is a mismatch between these traits and modern expectations, not something inherently “broken” in the brain.
No worries, since I’m sure we’ll all be back there soon enough and we’ll see how well the ADHDers fare.